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Astrocytes in (Patho)Physiology of the Nervous System
Title
Astrocytes in (Patho)Physiology of the Nervous System [electronic resource] / edited by Philip G. Haydon, Vladimir Parpura.
ISBN
9780387794921
Publication
Boston, MA : Springer US, 2009.
Physical Description
1 online resource (XX, 738 p.) 165 illus., 46 illus. in color.
Local Notes
Access is available to the Yale community.
Access and use
Access restricted by licensing agreement.
Summary
Due to their lack of electrical excitability, astrocytes, a subtype of glial cell, have long been neglected as active participants in intercellular communication within the central nervous system. Astrocytes, however, possess a diverse assortment of ion channels, neurotransmitter receptors, and transport mechanisms that enable them to respond to many of the same signals that act on neurons. Astrocytes in (Patho)Physiology of the Nervous System provides readers with a comprehensive description of the physiological roles astrocytes play in regulating neuronal activity and their critical involvement in pathophysiological states of the nervous system, including gliomas, Alexander's disease, and epilepsy. This book will be particularly useful for researchers, students, and workers in the field of neurobiology and cell biology. About the authors: Vladimir Parpura, M.D., Ph.D holds both a medical degree, awarded from the University of Zagreb in Croatia in 1989, and a doctorate, received in Neuroscience and Zoology from Iowa State University in 1993. He has held faculty appointments at the Department of Zoology and Genetics, Iowa State University and the Department of Cell Biology and Neuroscience, University of California Riverside. He is presently an Associate Professor in the Department of Neurobiology, University of Alabama Birmingham. His current research focuses on understanding the modulation of calcium-dependent glutamate release from astrocytes. Philip G. Haydon, Ph.D received his doctorate from the University of Leeds, England in 1982. He has held faculty appointments at the Department of Zoology and Genetics, Iowa State University, the Department of Neuroscience at the University of Pennsylvania, and has recently moved to Tufts University School of Medicine as Professor and Chair of the Department of Neuroscience. His research focuses on the role of astrocytes in the regulation of synapses, neuronal networks and behavior as well as how these glial cells contribute to neurological disorders.
Variant and related titles
Springer ENIN.
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Format
Books / Online
Language
English
Added to Catalog
April 05, 2019
Contents
Astrocyte Heterogeneity or Homogeneity?
Neural Stem Cells Disguised as Astrocytes
Neurotransmitter Receptors in Astrocytes
Specialized Neurotransmitter Transporters in Astrocytes
Connexin Expression (Gap Junctions and Hemichannels) in Astrocytes
Regulation of potassium by glial cells in the centralnervous system
Energy and Amino Acid Neurotransmitter Metabolism in Astrocytes
Calcium ion signaling in astrocytes
Astrocytes in Control of the Biophysical Properties of Extracellular Space
Structural association of astrocytes with neurons and vasculature: Defining territorial boundaries
Synaptic Information Processing by Astrocytes
Mechanisms of transmitter release from astrocytes
Release of Trophic Factors and Immune Molecules from Astrocytes
Molecular approaches for studying astrocytes
The tripartite synapse
Glia-derived D-serine and synaptic plasticity
Purinergic signaling in astrocyte function and interactions with neurons
Astrocyte control of blood flow
A Role for Glial Cells of the Neuroendocrine Brain in the Central Control of Female Sexual Development
Physiological and Pathological Roles of Astrocyte-mediated Neuronal Synchrony
Role of Ion Channels and Amino-Acid Transporters in the Biology of Astrocytic Tumors
Connexins and pannexins: Two gap junction families mediating glioma growth control
The Impact of Astrocyte Mitochondrial Metabolism on Neuroprotection During Aging
Alexander Disease: A Genetic Disorder of Astrocytes
Role of Astrocytes in Epilepsy
Hepatic Encephalopathy: A Primary Astrocytopathy.
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